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In these studies, the implicit mechanistic conclusion is that whenthe N20 response is absent, the anoxic injury experienced has been of sufficient sever-ity to destroy the primary somatosensory cortico-neuronal pool (or the thalamic relaynuclei projecting to that pool).

This diagram depicts two follicular cells: one in the process of thyro-globulin synthesis (on theleftwith redpathways) and the other in the process ofthyroglobulin resorption {on therightwith bluepathways).The numbers,which are described more fully in the text, indicate the sequential steps that occur: I, synthesis and secretion of thyroglobulin; 2, uptake and concen-tration of iodide from the blood by sodium/iodide symporters (NIS), release of iodide into the colloid via iodide/chloride (pendrin) transporters, andoxidation of iodide to iodine by thyroid peroxidase; 3, iodination of thyroglobulin in the colloid; 4, formation ofT3and T4 hormones in the colloid byoxidative coupling reactions; 5i, resorption of colloid via lysosomal pathway (major pathway); 5TE, resorption ofcolloid via megalin receptor-mediatedtransepithelial pathway; and 6, release ofT4 and T3from the cell into the circulation. magnetic resonance imaging and labelled leucocytescanning

magnetic resonance imaging and labelled leucocytescanning. The preparation is designed tostain the gray matter that is surrounded by the ascendingand descending nerve fibers.

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Moreover,this perspective seems to bracket out the significance given to accounting for deterioration insocial competence, and specific mental health conditions which is the more usual focus of mentalhealth as we age. The OI signi? -cantly decreased ( p= 0.05) from a baseline value in thesupine position of 15.7 ± 1.7–13.6 ± 1.6 after 1 h in theprone position and continued to decrease signi? cantly( p= 0.008) to 10.9 ± 1.5 after 19 h in the prone position.The OI at hour 21 in the supine position (14 ± 1.9) was notsigni?cantly different from that at baseline.